3 edition of Estimates of pulmonary and gastrointestinal deposition for occupational fiber exposures found in the catalog.
Estimates of pulmonary and gastrointestinal deposition for occupational fiber exposures
John M. Dement
by U.S. Dept. of Health, Education, and Welfare, Public Health Service, Center for Disease Control, National Institute for Occupational Safety and Health, Division of Surveillance, Hazard Evaluations, and Field Studies in Cincinnati, OH
Written in English
|Statement||John M. Dement, Robert L. Harris.|
|Series||NIOSH technical report, DHEW (NIOSH) publication -- no. 79-135., DHEW publication -- no. (NIOSH) 79-135.|
|Contributions||Harris, Robert L.|
|The Physical Object|
|Pagination||vi, 75 p. :|
|Number of Pages||75|
Formaldehyde exposure, acute pulmonary response, and exposure control options were evaluated in a group of 34 workers in a gross anatomy laboratory. Time-weighted average (TWA) exposure to formaldehyde ranged from – . The airborne fibers had a median length of pn and a median diameter of pm. The pulmonary deposition of fibers was measured 5 days after the last day of exposure; this period was allowed for clearance of approximately 95 percent of fibers deposited in the ciliated airways. Fiber deposition of the entire lung was percent.
Asthma is a disease of diffuse airway inflammation caused by a variety of triggering stimuli resulting in partially or completely reversible bronchoconstriction. Symptoms and signs include dyspnea, chest tightness, cough, and wheezing. The diagnosis is based on history, physical examination, and pulmonary function tests. Treatment involves. Air pollution, mainly from combustion, is one of the leading global health risk factors. A susceptible group is the more than million people worldwide suffering from chronic obstructive pulmonary disease (COPD). There are few data on lung deposition of airborne particles in patients with COPD and none for combustion particles. To determine respiratory Cited by:
Chronic Obstructive Pulmonary Disease is projected to rank 5th among the leading causes of disease by the year Risk factors include smoking, indoor and outdoor air pollution, previous respiratory infections, genetics, as well as occupational exposures. Aim The aim of this study was to determine the contribution of occupational exposures. model of the deposition and clearance kinetics of inhaled particles has considerable implications for occupational epidemiology, since anyexamination of data for the relation between dust exposure, dose received bythe lung, andthe incidence ofrespiratory disease will be influenced by the choice ofmodel of deposition and clearance. Exercises Cited by:
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Get this from a library. Estimates of pulmonary and gastrointestinal deposition for occupational fiber exposures. [John M Dement; Robert L Harris]. Dement, J.M., Harris, R.C. () Estimates of pulmonary and gastrointestinal deposition for occupational fiber exposures.
DHEW (NIOSH) Publication No. 79– (US Dept. of Health, Education and Welfare, National Institute for Occupational Safety and Health, Cincinnati, Ohio). Google ScholarCited by: 1.
A meta description is an HTML tag in the HTML code of your website, which allows you to customize a section of text that describes the page itself. It plays a role in how your page is seen by search engine crawlers, and how it appears in SERPs. Deposition was measured as a function of fiber length, diameter, and flow rate through the airways.
The overall deposition of the aerosols in the nasal airways ranged from 10 to 90 percent. The deposition increased with flow rate and was also somewhat higher with nasal hair stimulant in the anterior vestibule. Abstract. Inhalation of asbestos fibers results in a variety of neoplastic and nonneoplastic diseases of the respiratory tract.
Some of these diseases, such as asbestosis, generally occur after prolonged and intensive exposure to asbestos, whereas others, such as pleural mesothelioma, may occur following brief by: Answers is the place to go to get the answers you need and to ask the questions you want 'Estimates of pulmonary and gastrointestinal deposition for occupational fiber exposures' -.
BRIEF EPIDEMIOLOGY OF LUNG CANCER. Although lung cancer incidence rates started to slowly decrease for men in the s followed by declining incidence rates for women in the late s, 1 lung and bronchus cancer remain the leading cause of cancer mortality in the United States, with an estima deaths predicted to occur in men and women, Cited by: What authority does an Occupational Health and Safety Officer have if a cafe doesn't 'Estimates of pulmonary and gastrointestinal deposition for occupational fiber exposures' -.
ell formation. The mediastinal lymphadenopathy and the pulmonary infiltrates resolved after cessation of fiberglass exposure. The effects of fiberglass exposure on the respiratory system have been evaluated in several epidemiologic studies. Although fiberglass is widely used and low levels of exposure are commonplace, these studies have not shown a significant increase in.
(assuming 8 hrs/day, 5 days/wk, 50 wks/yr). The Yeh-Schum deposition model within MPPD was selected, along with the default model parameters, except for the following, which represent occupational exposures: breathing frequency ( breaths/min)  and tidal volume ( ml, to yield a minute volume of 20 L/min) .
Estimates of Pulmonary and Gastrointestinal Deposition for Occupational Fiber Exposures. DHEW publication No. 79– National Institute of Cited by: Uncontrolled occupational exposure to 1,1-dichloroFluoroethane (HCFCb) is associated with acute pulmonary toxicity.
Chest ; King MS, Eisenberg R, Newman JH, et al. Constrictive bronchiolitis in soldiers returning from Iraq and Afghanistan. Occupational Health Surveillance: Pulmonary Function Test in Proppant Occupational Diseases and Environme ntal Medicine,4, Published Online May i n S ciR es.
Occupational diesel exhaust exposure as a risk factor for chronic obstructive pulmonary disease Article Literature Review in Current opinion in pulmonary medicine 18(2).
Radon may be second only to smoking as a cause of lung cancer. The combination of smoking and radon exposure results in an especially serious health risk. The risk of lung cancer due to indoor radon exposure can be decreased with current technology.
This monograph is. OBJECTIVES Consolidation of epidemiological data on pancreatic cancer and worksite exposures. METHODS Publications during –98 were surveyed.
Studies without verified exposures were excluded. Meta-analyses were conducted on data from 92 studies covering populations, with results for 23 agents or groups of agents. With a standard format, five epidemiologists extracted risk estimates Cited by: Background Occupational exposures are associated with chronic obstructive pulmonary disease (COPD).
This study investigated this association among a population with a high prevalence of tuberculosis and smoking. Methods Cases (n=) diagnosed by pulmonologists were selected from specialist respiratory clinics. Frequency sex- and age Cited by: Unfortunately, this book can't be printed from the OpenBook.
If you need to print pages from this book, we recommend downloading it as a PDF. Visit to get more information about this book, to buy it in print, or to download it as a free PDF.
sampling over a year period were used to analyze forced expiratory volume in 1 second (FEV1) and FEV1/forced vital capacity (FVC). The exposure responses for declining pulmonary function and for possible early onset of BO were estimated using multiple regression methods.
Several exposure metrics were investigated; benchmark dose and excess lifetime risk of. Gulati, M.R. Cullen, in International Encyclopedia of Public Health, Asbestos Lung and Pleura Effects.
Asbestos is a strong incombustible fiber, which is used for fireproofing and insulation and comprises several forms including chrysotile, amosite, crocidolite, and rd workers, electricians, welders, and plumbers all may be potentially exposed.
– Idiopathic pulmonary fibrosis (lung scarring of unknown cause) – Lung cancer – Several other lung conditions (chronic infection, collagen‐ vascular disease, etc.) Can usually make right diagnosis with detailed history (occupational & medical) or, rarely, a lung biopsy.
Abstract. Adverse health effects of inhaled particulates have been noted for centuries, and studies over the last several decades have linked particle and fiber inhalation to several pulmonary diseases, including fibrosis, silicosis, chronic obstructive pulmonary disease, emphysema, asthma, hypersensitivity pneumonitis, and cancer, as well as cardiovascular Cited by: 3.Patients with lung disease suspected to be due to exposure to occupational or environmental agents can be evaluated at the UCSF Occupational and Environmental Medicine Practice at Mt.
Zion. The practitioners there can consult with board-certified Occupational Medicine physicians, as needed, and utilize additional assessments, including pulmonary function testing, sleep .